By A. Revhaug (auth.), Professor Arthur Revhaug (eds.)
This objective of this ebook is to target a really universal scenario obvious in clinical perform, the intense catabolic country. This pathophysiological take a seat uation is never mentioned as a separate entity, most likely since it is visible in all specialties of drugs and effects from an excellent range of brokers, insults, and illnesses. It hence excessive time to assemble an important and up to date wisdom in this entity, and the first target of the publication is to provide a set of up to date info at the acute catabolic country. one other goal of the booklet is to clarify that, except the overall reaction of the complete organism through the acute catabolic country, a chain of organ-specific responses also will occur, which also needs to be thought of in the course of remedy. It has develop into very away from those organ-spe from engaged on this ebook that present wisdom cific responses is especially sparse and in a few parts nearly nonexistent; this publication hence additionally makes a speciality of the responses and alterations which occur in several organs throughout the acute catabolic nation and the inter motion among those organs and their responses.
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28 N. N. Abumrad and P. E. Molina Of the three main gluconeogenic substrates, lactate derived from the periphery, most likely as the result of increased muscle glycogenolysis, appears to account for most of the gluconeogenic precursor supply . Although the increase in glycerol uptake by the liver in response to glucopenia is significant, the total amount of three carbon precursors that lactate contributes is significantly greater. Although it could be argued that the efficiency with which the liver converts glycerol and alanine into glucose is greater than that for lactate, we have provided evidence for a critical contribution oflactate as a gluconeogenic precursor.