By Prof. Dr. Wolfgang Dahr, John Moulds, Phyllis Unger, Dominique Blanchard (auth.), Prof. Dr. med. B. Brinkmann, Dr. med. Klavs Henningsen (eds.)
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Additional resources for 11th Congress of the Society for Forensic Haemogenetics (Gesellschaft für forensische Blutgruppenkunde e.V.): Copenhagen, August 6–10, 1985
Estimates derived from electrophoretic studies indicate that approximately one third of all human enzymes exhibit genetic polymorphism where "polymorphism" is defined as the occurrence of heterozygotes with a frequency greater than 2%. 06 and this implies that any single individual in any human population is likely to be heterozygous at about 6% of the loci encoding enzyme proteins, for alleles which give rise to electrophoretically distinct isozyme forms (Harris & Hopkinson, 1976). The complexity of the isozyme patterns varies according to the subunit structure of the enzyme proteins and on average monomeric enzymes exhibit a higher incidence of genetic polymorphism than multimeric enzymes (Harris, Hopkinson & Edwards, 1977).
The Class II molecules are properdin factor Bf of the alternative and factor 2 (C2) and 4 (C4) of the classical complement activation pathway. All Class I, II and III molecules are genetically highly polymorphic: there is a large number of different HLA types in the population, but all HLA factors show pronounced linkage disequilibrium with at least one other factor controlled by genes at a nearby HLA locus. The Class I and II molecules are intimately involved in the specific thymusdependent immune response.
J. Clin. Chemistry, 45, Suppl. : The genetic markers of human immunoglobulins. : Stimuli for anti-Ig-allotypic antibodies with particular reference to rheumatoid arthritis. Clin. Immun. : Burkitt lymphoma and genetic markers of immunoglobulin. Clin. Exp. Immunogen. : Immunoglobulin genes. Ann. Rev. Immunol. : Typing of genetic variants of a 1-antitrypsin by electrofocusing. Clin. Chem. : Recurrent idiotopes and internal images. EMBO Journ. : A Bi-directional immune network mechanism: Simultaneous induction of idiotype and antianti-idiotype in rabbits immunized with antibody to a common idiotypic specificity of anti-VHa2-allotype antibodies.